Vitamin Deficiency Linked to Stroke and Plaque Buildup in Carotid Artery

Vitamin Deficiency Linked to Stroke and Plaque Buildup in Carotid Arteries

Ronald Grisanti D.C., D.A.B.C.O., D.A.C.B.N., MS

A study published in the the Canadian Medical Association Journal revealed that people low in vitamin B12 had an increase risk of a fatal heart attack and stroke.

The study focused on the relationship between homocysteine, B-12 and carotid artery plaque.

The study showed that higher blood levels of B vitamins are related to lower concentrations of homocysteine leading to decrease plaquing in the carotid arteries. However, an elevated blood homocysteine level revealed a strong risk factor for heart disease and stroke.

How the Study was Conducted
The study examined 421 people with the average age being 66. Vitamin B12, homocysteine levels and degree of plaque in the carotid arteries (via ultrasound) were evaluated.

The Results

Seventy-three patients (17%) had vitamin B12 deficiency with significant elevation of homocysteine. In addition and most important, carotid plaque was significantly larger among the group of patients who had deficiency of vitamin B12 In conclusion, the authors found that low blood vitamin B12 levels are a major cause of elevated homocysteine levels and increased carotid plaque area.

Dr. Grisanti's Comments
Have your physician order a blood homocysteine test and a methylmalonic acid (MMA) test. This is the most specific test for B12 status NOT the serum B-12 blood test.


Reference

Robertson J, Iemolo F, Stabler SP, Allen RH, Spence JD. Vitamin B12, homocysteine and carotid plaque in the era of folic acid fortification of enriched cereal grain products. CMAJ. 2005 Jun 7;172(12):1569-73.

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Medications That Contribute to Alzheimer's and Natural Ways to Improve Memory

Alzheimer's: Why is the Brain Deteriorating?

Ronald Grisanti D.C., D.A.B.C.O., D.A.C.B.N., M.S.

After considerable research it is interesting to bring you up to speed on documented evidence of things which answer the question. "Why is the human brain deteriorating faster than the rest of the body?”

There are a multitude of factors and today's article will touch on a few and also provide some solutions.

For starters I find it disturbing and somewhat criminal that a common blood pressure medication called calcium channel blockers has been proven radiologically on MRI to cause brain shrinking. Research has shown that these drugs cause deterioration of the I.Q. within 5 years' use.

Another medication used to lower cholesterol called Lipitor causes a decline in brain function. It is important to know that statin cholesterol-lowering drugs like Lipitor poison the liver's synthesis of cholesterol. This in turn will starve the brain of cholesterol needed to repair the brain, renew worn out membranes, and stave off Alzheimer's.

In fact, an excellent book, “Lipitor Thief of Memory” written by the respected medical doctor, former astronaut, aerospace medical research scientist, flight surgeon, and family doctor, Dr. Duane Graveline, shares his rapid mental decline after taking the drug Lipitor. Worth reading.

Even with all this hard evidence can you believe the pharmaceutical industry has created a potent drug which combines both the calcium channel blocker and a statin called Atorvastatin/Amlodipine (Caduet). Talk about a double punch to optimal brain function!

Moving on to another documented contributor of Alzheimer's, we can't forget the unavoidable heavy metals. We all have them in us and they poison brain repair enzymes, leading to Alzheimer's.

For example, there is no one who doesn't have aluminum in them, from eating out, aluminum cookware, aluminum flocculation agents in municipal drinking waters, aluminum in baking powders used in breads, processed and restaurant foods cooked in aluminum vats, industrial and vehicular exhausts, deodorants, antacids, and many other sources.

Aluminum causes the nerves in the brain to actually get tangled up (neurofibrillary tangles) as well as make a glue-like substance (called amyloid) to gum up the normal workings of the delicate brain electricity..

Now to provide some nutritional answers to reduce amyloid production we need to look no further than Phosphatidylserine (PS). This nutritional powerhouse has shown to perk up memory, and stave off Alzheimer's.  One interested case showed PS in 3 months return the memory back to where it was 12 years earlier.

Most recently there has been evidence how DHA is an amyloid eater.

Well here is something even easier: green tea. Real organic green tea has over 3 catechins or polyphenols. They have been found to be potent preventers of amyloid deposition in the brain. Sencha Premium Organic Green Tea is by far the best I have found.

This short article is simply a glimpse of the research you won't see promoted on CNN or Fox News. Of course this is sad. There is another side of the clinical management of many diseases that the public will rarely if ever be shown unless you are a reader of my weekly health reports or other alternative or functionally oriented heath professional reports or journals.

The take away from today's article is to “NOT” be your own doctor but seek out the assistance and help from someone trained and skilled in functional medicine who can properly evaluate you and outline a personalized program to help you get well.  

To find a healthcare professional certified in functional medicine, go to www.FunctionalMedicineDoctors.com.These are clinicians who have been trained at Functional Medicine University (www.FunctionalMedicineUniversity.com)

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Reference

Haque A, et al, Green tea catechins prevent cognitive deficits caused by AB1-40 in rats, J Nutr Biochem, 19:619-26, 2008

Behl C, et al, Vitamin E protects nerve cells from amyloid B protein toxicity, Biochem Biophys Res Commun, 186:944-52, 1992

Hashimoto M, et al, Docosahexaenoic acid provides protection from impairment of learning ability in Alzheimer's disease model rats, J Neurochem, 81:1084-91, 2002

Rezai-Zadeh K, et al, Green tea epigallocatechin-3-gallate (EGCG) modulates amyloid precursor protein cleavage and reduces cerebral amyloidosis in Alzheimer transgenic mice, J Neurosci, 25:8807-14, 2005

Crook T1, Petrie W, Wells C, Massari DC.Effects of phosphatidylserine in Alzheimer's disease.Psychopharmacol Bull. 1992;28(1):61-6.

http://www3.scienceblog.com/community/older/1997/B/199702039.html (Calcium Channel Blocker reference)

 

Alzheimer's or Brain Diabetes

Alzheimer's or Brain Diabetes?

Ronald Grisanti D.C., D.A.B.C.O., D.A.C.B.N., M.S.

 

A growing body of research suggests there's a powerful connection between your diet and your risk of Alzheimer's disease via similar pathways that cause type 2 diabetes.

Back in 2005, a published medical paper introduced a new disease tentatively dubbed "type 3 diabetes".

The researchers learned that, in addition to the pancreas, your brain also produces insulin.

They discovered that without the insulin your brain cells will die.

A drop in insulin production in your brain may contribute to the degeneration of your brain cells, and studies have found that people with lower levels of insulin and insulin receptors in their brain often have Alzheimer's disease.

Studies since 2005 have continually documented that insulin has a much greater role in the brain than previously expected.

Insulin is directly responsible for neuron glucose-uptake, and the regulation of neurotransmitters like acetylcholine, which are crucial for memory and learning.

Scientists have come to understand that cognition is impaired when insulin levels are reduced.

The clinical research has made it quite clear that the same pathological process that leads to insulin resistance and type 2 diabetes may also hold true for your brain.

The take away from these studies make a strong point that the over-consumption of sugars and "grains" which are also detrimental to the the development of diabetes may also result in Type 3 Diabetes (brain diabetes).

When the brain becomes overwhelmed by the consistently high levels of glucose, the insulin signaling will eventually become blunted or desensitized. This will lead to impairments in your thinking and memory abilities, eventually causing permanent brain damage.

Healthcare clinicians trained in functional medicine have the training and knowledge to investigate what is at the root of the pathological process that leads to Type 2 diabetes and the new diagnosis of Type 3 diabetes.

The one size fits all approach of prescriptive medications for diabetes, although of some value, will not shut down the physiological cascade of the consequences of poor sugar/insulin control.


Reference:

Steen E, Terry BM, Rivera EJ, Cannon JL, Neely TR, Tavares R, Xu XJ, Wands JR, de la Monte SM. Impaired insulin and insulin-like growth factor expression and signaling mechanisms in Alzheimer's disease--is this type 3 diabetes? J Alzheimers Dis. 2005 Feb;7(1):63-80.

de la Monte, Wands.Alzheimer's Disease Is Type 3 Diabetes–Evidence Reviewed, J Diabetes Sci Technol. 2008 Nov; 2(6): 1101–1113.

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Link Between Blood Pressure and Sunlight

Functional Medicine University

Lower Blood Pressure: Surprising New Study

Ronald Grisanti D.C., D.A.B.C.O., D.A.C.B.N., M.S.

There is now a new natural weapon to combat against the growing population of high blood pressure sufferers.

 

 

Now this new weapon is as close as your backyard.

What I am talking about is good old sunlight.

Blood pressure levels are commonly higher during winter months.

The question you may ask is what is the mechanism that allows sunlight to lower blood pressure?

British researchers have figured out why.

The answer is nitric oxide (NO).

Nitric oxide is known to reduce blood pressure by evoking vasodilation either directly by causing relaxation of vascular smooth muscle or indirectly by acting in the rostral brainstem to reduce central sympathetic outflow, which decreases the release of norepinephrine from sympathetic nerve terminals.

Basically, nitric oxide increases the elasticity of the artery walls and helps to normalize high blood pressure.

An increasingly large body of literature suggests that alterations in the NO system may play an important role in the development or maintenance of clinical hypertension. 

What they found is that nitric oxide stored in the top layers of the skin reacts to sunlight and causes blood vessels to widen as the oxide moves into the bloodstream. That, in turn, lowers blood pressure.

According to researcher Martin Feelisch, a professor of experimental medicine and integrative biology at the University of Southampton, exposure to ultraviolet light might help reduce the risk for heart disease.

"This new study finds that UV light exposure to the skin induced nitric oxide release and modestly lowered blood pressure, suggesting that this may play a role in modulating blood pressure," said Fonarow, a spokesman for the American Heart Association.

In 2009, a team led by the University of Edinburgh's Richard Weller showed that human skin and the dermal vasculature contain significant stores of NO—much more than can be found circulating in the blood—and that these stores could be mobilized by UVA (long-wave UV) irradiation.

“This study provides suggestive evidence that skin-derived NO metabolites may have a role in modulation of blood pressure upon UV exposure,” Thomas Michel, a professor of medicine and biochemistry at Harvard Medical School.


 

 


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Reference:

Donald Liu, Bernadette O Fernandez, Alistair Hamilton, Ninian N Lang, Julie M C Gallagher, David E Newby, Martin Feelisch and Richard B Weller, UVA Irradiation of Human Skin Vasodilates Arterial Vasculature and Lowers Blood Pressure

Taking It To The Next Level

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Gratitude

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